A possible mechanism for increments of calcium and protein kinase C activity in kidney of experimental acute fluorosis.
Suketa
Y
Y
Department of Environmental Biochemistry and Toxicology, University of Shizuoka School of Pharmaceutical Sciences, Japan.
Sakai
Y
Y
D000818:Animals; D002118:Calcium / Q000378:metabolism*; D000242:Cyclic AMP / Q000097:blood; D005459:Fluorides / Q000494:pharmacology; D009050:Fluorosis, Dental / Q000378:metabolism*; D007668:Kidney / Q000187:drug effects / Q000201:enzymology*; D008297:Male; D009543:Nifedipine / Q000494:pharmacology; D019269:Phosphatidylinositol 4,5-Diphosphate; D010716:Phosphatidylinositols / Q000378:metabolism; D011493:Protein Kinase C / Q000378:metabolism*; D051381:Rats; D011919:Rats, Inbred Strains; D010738:Type C Phospholipases / Q000378:metabolism
D019269:Phosphatidylinositol 4,5-Diphosphate; D010716:Phosphatidylinositols; D000242:Cyclic AMP; D011493:Protein Kinase C; D010738:Type C Phospholipases; D009543:Nifedipine; D005459:Fluorides; D002118:Calcium