title: The increase in coronary flow induced by converting enzyme inhibitors is prostacyclin independent.
authors.affiliation: Department of Clinical Pharmacology, University of Gronigen, The Netherlands.
authors.forename: R A
authors.initials: RA
authors.lastname: Tio
authors.forename: J
authors.initials: J
authors.lastname: van Wijngaarden
authors.forename: E
authors.initials: E
authors.lastname: Scholtens
authors.forename: W H
authors.initials: WH
authors.lastname: van Gilst
authors.forename: C D
authors.initials: CD
authors.lastname: de Langen
authors.forename: H
authors.initials: H
authors.lastname: Wesseling
mesh_terms: D015121:6-Ketoprostaglandin F1 alpha / Q000096:biosynthesis; D000806:Angiotensin-Converting Enzyme Inhibitors / Q000494:pharmacology*; D000818:Animals; D001920:Bradykinin / Q000494:pharmacology; D002216:Captopril / Q000031:analogs & derivatives / Q000494:pharmacology; D003326:Coronary Circulation / Q000187:drug effects*; D015773:Enalaprilat / Q000494:pharmacology; D011464:Epoprostenol / Q000502:physiology*; D066298:In Vitro Techniques; D007213:Indomethacin / Q000494:pharmacology; D008297:Male; D051381:Rats; D011919:Rats, Inbred Strains
chemical_list: D000806:Angiotensin-Converting Enzyme Inhibitors; C044958:zofenopril; D015121:6-Ketoprostaglandin F1 alpha; D002216:Captopril; D011464:Epoprostenol; D015773:Enalaprilat; D001920:Bradykinin; D007213:Indomethacin