title: Campylobacter pylori and duodenal ulcers: the gastrin link.
abstract: The possibility that Campylobacter pylori (CP) in the gastric antrum stimulates gastrin release in duodenal ulcer (DU) disease was examined in 31 patients. The 25 patients with antral colonisation with CP had higher basal and meal-stimulated plasma gastrin concentrations, and higher peak acid output (PAO), than did the 6 without CP in the autumn.
authors.affiliation: Department of Medicine, Royal Postgraduate Medical School, Hammersmith Hospital, London.
authors.forename: S
authors.initials: S
authors.lastname: Levi
authors.forename: K
authors.initials: K
authors.lastname: Beardshall
authors.forename: G
authors.initials: G
authors.lastname: Haddad
authors.forename: R
authors.initials: R
authors.lastname: Playford
authors.forename: P
authors.initials: P
authors.lastname: Ghosh
authors.forename: J
authors.initials: J
authors.lastname: Calam
mesh_terms: D000328:Adult; D000368:Aged; D002169:Campylobacter Infections / Q000150:complications*; D004381:Duodenal Ulcer / Q000378:metabolism* / Q000382:microbiology; D004435:Eating; D005069:Evaluation Studies as Topic; D005260:Female; D005744:Gastric Acid / Q000378:metabolism*; D005745:Gastric Acidity Determination; D005755:Gastrins / Q000097:blood / Q000378:metabolism*; D006801:Humans; D008297:Male; D008875:Middle Aged; D010418:Pentagastrin; D011706:Pyloric Antrum / Q000382:microbiology
chemical_list: D005755:Gastrins; D010418:Pentagastrin