Antagonistic modulation of S-K+ channel activity by cyclic AMP and arachidonic acid metabolites. Role for two G proteins.
Volterra
A
A
Department of Pharmacology, Howard Hughes Medical Institute, Columbia University, New York, New York 10032.
Siegelbaum
S A
SA
D000200:Action Potentials / Q000187:drug effects; D000818:Animals; D001048:Aplysia; D001095:Arachidonic Acids / Q000502:physiology*; D000242:Cyclic AMP / Q000502:physiology*; D019835:FMRFamide; D019204:GTP-Binding Proteins / Q000502:physiology*; D066298:In Vitro Techniques; D008564:Membrane Potentials / Q000187:drug effects; D009046:Motor Neurons / Q000187:drug effects / Q000502:physiology*; D009475:Neurons, Afferent / Q000187:drug effects / Q000502:physiology*; D009479:Neuropeptides / Q000494:pharmacology; D015221:Potassium Channels / Q000502:physiology*; D011494:Protein Kinases / Q000378:metabolism; D015290:Second Messenger Systems; D012701:Serotonin / Q000494:pharmacology
D001095:Arachidonic Acids; D009479:Neuropeptides; D015221:Potassium Channels; D012701:Serotonin; D019835:FMRFamide; D000242:Cyclic AMP; D011494:Protein Kinases; D019204:GTP-Binding Proteins