Excitotoxins: a possible new mechanism for the pathogenesis of ischemic retinal damage.
Bresnick
G H
GH
Department of Ophthalmology, University of Wisconsin Medical School, Madison 53792.
D000328:Adult; D000818:Animals; D001224:Aspartic Acid / Q000378:metabolism / Q000502:physiology; D002415:Cats; D002478:Cells, Cultured; D005971:Glutamates / Q000378:metabolism / Q000502:physiology; D006801:Humans; D007511:Ischemia / Q000209:etiology*; D051379:Mice; D009474:Neurons / Q000187:drug effects; D009498:Neurotoxins / Q000037:antagonists & inhibitors / Q000502:physiology*; D018377:Neurotransmitter Agents / Q000037:antagonists & inhibitors / Q000502:physiology; D010786:Photoreceptor Cells / Q000378:metabolism; D011817:Rabbits; D051381:Rats; D012164:Retinal Diseases / Q000209:etiology; D012171:Retinal Vessels
D005971:Glutamates; D009498:Neurotoxins; D018377:Neurotransmitter Agents; D001224:Aspartic Acid