[Short term effects of carbon disulfide on the intracardial irritation development and transmission in the rat].
Studies were conducted into effects of acute and subacute CS2 application on intracardiac irritability and conduction in rat. Aconitine-induced arrhythmia and the coronary occlusion method were used as pathophysiological models. Under physiological conditions, short-time exposure to CS2 caused deceleration of intracardiac impulse conduction, while under pathophysiological conditions, modified arrhythmia or reduced survival rate was the result.
Hoffmann
P
P
Klapperstück
M
M
Grimm
I
I
Böhme
K
K
D000818:Animals; D001145:Arrhythmias, Cardiac / Q000503:physiopathology*; D002246:Carbon Disulfide / Q000633:toxicity*; D003327:Coronary Disease / Q000503:physiopathology*; D004195:Disease Models, Animal; D004562:Electrocardiography; D006321:Heart / Q000187:drug effects*; D006339:Heart Rate / Q000187:drug effects; D008297:Male; D051381:Rats
D002246:Carbon Disulfide
Kurzzeiteffekte von Schwefelkohlenstoff auf die intrakardiale Reizbildung und Reizbildung und Reizleitung bei der Ratte.