[Prostaglandins and chronic proliferative glomerulonephritis].
In 14 patients with chronic proliferative glomerulonephritis, corrected arterial hypertension and normal or marginal glomerular filtration the authors assessed plasmatic and urinary metabolites of PGI2 and TXA2. They found that the production of both PGI2 and TXA2 was raised in the organism and they assume that in the stimulated synthesis hypertension and its treatment participated. The production of both prostaglandins in the kidneys was, however, normal.
Petrů
I
I
Knotková
V
V
Nĕmecek
K
K
Pick
P
P
Peleska
J
J
Jáchymová
M
M
Tomasy
M
M
D015121:6-Ketoprostaglandin F1 alpha / Q000378:metabolism; D002908:Chronic Disease; D011464:Epoprostenol / Q000378:metabolism; D005260:Female; D015432:Glomerulonephritis, Membranoproliferative / Q000378:metabolism*; D006801:Humans; D008297:Male; D011453:Prostaglandins / Q000378:metabolism*; D013928:Thromboxane A2 / Q000378:metabolism; D013929:Thromboxane B2 / Q000378:metabolism
D011453:Prostaglandins; D013929:Thromboxane B2; D013928:Thromboxane A2; D015121:6-Ketoprostaglandin F1 alpha; D011464:Epoprostenol
Prostaglandiny a chronická proliferativní glomerulonefritida.