Is activated C3 a premier factor of DIC development in septic shock?
Wu
Q X
QX
Department of Pathophysiology, Peking Union Medical College, Beijing, China.
Liu
Z Y
ZY
Dang
Y
Y
Chen
L
L
Chen
H C
HC
D000110:Acetylcholinesterase / Q000097:blood; D000706:Anaphylatoxins; D000818:Animals; D001792:Blood Platelets / Q000378:metabolism; D003176:Complement C3 / Q000378:metabolism / Q000494:pharmacology / Q000502:physiology*; D015926:Complement C3a; D004211:Disseminated Intravascular Coagulation / Q000097:blood / Q000209:etiology*; D004731:Endotoxins / Q000494:pharmacology; D004926:Escherichia coli; D005966:Glucuronidase / Q000097:blood; D006801:Humans; D008585:Meningitis, Meningococcal / Q000097:blood / Q000150:complications*; D010976:Platelet Count; D011817:Rabbits; D012701:Serotonin / Q000097:blood; D012772:Shock, Septic / Q000097:blood / Q000150:complications*
D000706:Anaphylatoxins; D003176:Complement C3; D004731:Endotoxins; D012701:Serotonin; D015926:Complement C3a; D000110:Acetylcholinesterase; D005966:Glucuronidase