title: Is activated C3 a premier factor of DIC development in septic shock?
authors.affiliation: Department of Pathophysiology, Peking Union Medical College, Beijing, China.
authors.forename: Q X
authors.initials: QX
authors.lastname: Wu
authors.forename: Z Y
authors.initials: ZY
authors.lastname: Liu
authors.forename: Y
authors.initials: Y
authors.lastname: Dang
authors.forename: L
authors.initials: L
authors.lastname: Chen
authors.forename: H C
authors.initials: HC
authors.lastname: Chen
mesh_terms: D000110:Acetylcholinesterase / Q000097:blood; D000706:Anaphylatoxins; D000818:Animals; D001792:Blood Platelets / Q000378:metabolism; D003176:Complement C3 / Q000378:metabolism / Q000494:pharmacology / Q000502:physiology*; D015926:Complement C3a; D004211:Disseminated Intravascular Coagulation / Q000097:blood / Q000209:etiology*; D004731:Endotoxins / Q000494:pharmacology; D004926:Escherichia coli; D005966:Glucuronidase / Q000097:blood; D006801:Humans; D008585:Meningitis, Meningococcal / Q000097:blood / Q000150:complications*; D010976:Platelet Count; D011817:Rabbits; D012701:Serotonin / Q000097:blood; D012772:Shock, Septic / Q000097:blood / Q000150:complications*
chemical_list: D000706:Anaphylatoxins; D003176:Complement C3; D004731:Endotoxins; D012701:Serotonin; D015926:Complement C3a; D000110:Acetylcholinesterase; D005966:Glucuronidase