title: Leg blood flow during total hip replacement under spinal or general anaesthesia.
abstract: Calf blood flow was studied using venous occlusion impedance plethysmography during 122 total hip arthroplasties. Patients were randomly allocated to receive spinal or general anaesthesia. Blood flow was measured nine times perioperatively. In the non-surgical leg, mean blood flow rose by over 50% in both groups following anaesthetic induction, remaining significantly elevated with spinal but falling back gradually to baseline with general anaesthesia. In the surgical leg, surgical manipulations produced marked falls in flow in many patients, particularly with femoral component insertion. If this occurred, hyperaemia was commonly seen with spinal anaesthesia but rarely with general anaesthesia once the joint was relocated. Venous outflow resistance rose slightly during anaesthesia in both groups, more so with general anaesthesia. In the surgical leg, marked rises occurred with surgical manipulations, but resistance fell abruptly once the joint was relocated. No clear relationship between these observations and the occurrence of deep vein thrombosis postoperatively was established.
authors.affiliation: Division of Anaesthesia, Christchurch Clinical School of Medicine, University of Otago, New Zealand.
authors.forename: F M
authors.initials: FM
authors.lastname: Davis
authors.forename: V G
authors.initials: VG
authors.lastname: Laurenson
authors.forename: W J
authors.initials: WJ
authors.lastname: Gillespie
authors.forename: J
authors.initials: J
authors.lastname: Foate
authors.forename: A D
authors.initials: AD
authors.lastname: Seagar
mesh_terms: D000768:Anesthesia, General; D000775:Anesthesia, Spinal; D001783:Blood Flow Velocity; D005260:Female; D006621:Hip Joint / Q000601:surgery; D006622:Hip Prosthesis; D006801:Humans; D007866:Leg / Q000098:blood supply*; D008297:Male; D010992:Plethysmography, Impedance; D011183:Postoperative Complications / Q000503:physiopathology; D011897:Random Allocation; D013924:Thrombophlebitis / Q000209:etiology / Q000503:physiopathology