Does clonidine- or neuropeptide Y-mediated inhibition of ATP secretion from sympathetic nerves operate primarily by increasing a potassium conductance?
Department of Physiology, Karolinska Institutet, Stockholm, Sweden.
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D000255:Adenosine Triphosphate / Q000378:metabolism*; D000818:Animals; D003000:Clonidine / Q000494:pharmacology*; D008297:Male; D051379:Mice; D009478:Neuropeptide Y / Q000494:pharmacology*; D011188:Potassium / Q000502:physiology*; D015221:Potassium Channels / Q000502:physiology; D011467:Prostate / Q000294:innervation; D013564:Sympathetic Nervous System / Q000187:drug effects / Q000378:metabolism*; D014649:Vas Deferens / Q000294:innervation
D009478:Neuropeptide Y; D015221:Potassium Channels; D000255:Adenosine Triphosphate; D003000:Clonidine; D011188:Potassium