[Hyperreninemic hypoaldosteronism syndrome induced by plasma exchange].
The study was designed to measure sequential changes in plasma renin activity, aldosterone, angiotensin-converting enzyme activity and ionograms, prior to, and after therapeutic plasma exchange. Each measurement was repeated before and after stimulation of renin activity induced by furosemide. The results showed that plasma exchange induces a syndrome of hyperreninemic hypoaldosteronism associated with a depletion in angiotensin-converting enzyme activity which might account for the dissociation between plasma renin activity and aldosterone.
Fourrier
F
F
Service de Rénimation polyvalente, Hôpital B, Lille.
Leclerc
L
L
Racadot
A
A
Wemeau
J L
JL
Lestavel
P
P
Chopin
C
C
D000293:Adolescent; D000328:Adult; D000368:Aged; D005260:Female; D005665:Furosemide; D006801:Humans; D006994:Hypoaldosteronism / Q000097:blood / Q000209:etiology*; D008297:Male; D008875:Middle Aged; D007703:Peptidyl-Dipeptidase A / Q000097:blood; D010951:Plasma Exchange / Q000009:adverse effects* / Q000379:methods; D012083:Renin / Q000097:blood*; D012084:Renin-Angiotensin System / Q000187:drug effects; D013577:Syndrome; D013997:Time Factors
D005665:Furosemide; D007703:Peptidyl-Dipeptidase A; D012083:Renin
Syndrome d'hypoaldostéronisme hyperréninémique induit par les échanges plasmatiques.