Peptic ulcer disease: physiology and pathophysiology.
Holt
K M
KM
Isenberg
J I
JI
D000863:Antacids / Q000494:pharmacology / Q000627:therapeutic use; D001241:Aspirin / Q000009:adverse effects; D001729:Bismuth / Q000494:pharmacology / Q000627:therapeutic use; D004381:Duodenal Ulcer / Q000139:chemically induced / Q000188:drug therapy / Q000503:physiopathology; D005744:Gastric Acid / Q000378:metabolism; D005753:Gastric Mucosa / Q000378:metabolism / Q000502:physiology; D006635:Histamine H2 Antagonists / Q000627:therapeutic use; D006801:Humans; D006863:Hydrogen-Ion Concentration; D010276:Parasympatholytics / Q000494:pharmacology / Q000627:therapeutic use; D010295:Parietal Cells, Gastric / Q000502:physiology / Q000648:ultrastructure; D010435:Pepsinogens / Q000378:metabolism; D010437:Peptic Ulcer / Q000188:drug therapy / Q000503:physiopathology*; D011453:Prostaglandins / Q000494:pharmacology / Q000627:therapeutic use; D012907:Smoking; D013270:Stomach / Q000033:anatomy & histology / Q000502:physiology*; D013276:Stomach Ulcer / Q000139:chemically induced / Q000188:drug therapy / Q000503:physiopathology
D000863:Antacids; D006635:Histamine H2 Antagonists; D010276:Parasympatholytics; D010435:Pepsinogens; D011453:Prostaglandins; D001241:Aspirin; D001729:Bismuth