Limbic seizure and brain damage produced by kainic acid: mechanisms and relevance to human temporal lobe epilepsy.
Ben-Ari
Y
Y
D000818:Animals; D001665:Binding Sites; D001921:Brain / Q000378:metabolism; D002118:Calcium / Q000378:metabolism; D004195:Disease Models, Animal; D004347:Drug Interactions; D004833:Epilepsy, Temporal Lobe / Q000209:etiology*; D005947:Glucose / Q000378:metabolism; D005971:Glutamates / Q000494:pharmacology; D018698:Glutamic Acid; D006801:Humans; D007608:Kainic Acid / Q000378:metabolism / Q000494:pharmacology*; D008032:Limbic System / Q000187:drug effects* / Q000378:metabolism; D011759:Pyrrolidines / Q000494:pharmacology*; D013226:Status Epilepticus / Q000139:chemically induced; D015032:Zinc / Q000378:metabolism
D005971:Glutamates; D011759:Pyrrolidines; D018698:Glutamic Acid; D005947:Glucose; D015032:Zinc; D007608:Kainic Acid; D002118:Calcium