Symptomatic hemidystonia of delayed onset. Magnetic resonance demonstration of pathology in the putamen and the caudate nucleus.
We present a case of symptomatic hemidystonia of delayed onset. The primary disease was a perinatal, presumed cerebrovascular infarction brought about by febrile illness with convulsions 12 weeks after partus. After many years without neurological symptoms, the hemidystonia started in adolescence, and became stationary after 4 years of mild progression. Magnetic resonance imaging revealed atrophy of the right striatum including the caudate nucleus and putamen. The symptoms responded moderately to treatment with benzhexol.
Midgard
R
R
Department of Neurology, University of Bergen, Norway.
Aarli
J A
JA
Julsrud
O J
OJ
Odegaard
H
H
D000328:Adult; D001284:Atrophy; D001479:Basal Ganglia / Q000098:blood supply; D002421:Caudate Nucleus / Q000473:pathology*; D002544:Cerebral Infarction / Q000150:complications; D004421:Dystonia / Q000188:drug therapy / Q000209:etiology* / Q000503:physiopathology; D006801:Humans; D008279:Magnetic Resonance Imaging; D008297:Male; D011699:Putamen / Q000473:pathology*; D014282:Trihexyphenidyl / Q000627:therapeutic use
D014282:Trihexyphenidyl