Uterine glucose metabolism in the prepubertal rat treated neonatally with androgen, estrogen, and antihormones.
Estrogen secretion during infancy may selectively enhance the phosphogluconate oxidative pathway in the rat uterus, for altered estrogen-stimulated glucose oxidation prepubertally is correlated (+0.91) with impaired ovarian development and not uterine estrogen receptor content.
D000728:Androgens / Q000502:physiology*; D000818:Animals; D000831:Animals, Newborn / Q000502:physiology*; D003534:Cyproterone / Q000031:analogs & derivatives / Q000494:pharmacology; D017373:Cyproterone Acetate; D004958:Estradiol / Q000494:pharmacology; D004965:Estrogen Antagonists / Q000494:pharmacology; D004967:Estrogens / Q000502:physiology*; D005260:Female; D005947:Glucose / Q000378:metabolism*; D009929:Organ Size / Q000187:drug effects; D010053:Ovary / Q000033:anatomy & histology; D051381:Rats; D011919:Rats, Inbred Strains; D013739:Testosterone / Q000494:pharmacology; D014599:Uterus / Q000187:drug effects / Q000378:metabolism*
D000728:Androgens; D004965:Estrogen Antagonists; D004967:Estrogens; D013739:Testosterone; D017373:Cyproterone Acetate; D004958:Estradiol; D003534:Cyproterone; D005947:Glucose