title: Uterine glucose metabolism in the prepubertal rat treated neonatally with androgen, estrogen, and antihormones.
abstract: Estrogen secretion during infancy may selectively enhance the phosphogluconate oxidative pathway in the rat uterus, for altered estrogen-stimulated glucose oxidation prepubertally is correlated (+0.91) with impaired ovarian development and not uterine estrogen receptor content.
mesh_terms: D000728:Androgens / Q000502:physiology*; D000818:Animals; D000831:Animals, Newborn / Q000502:physiology*; D003534:Cyproterone / Q000031:analogs & derivatives / Q000494:pharmacology; D017373:Cyproterone Acetate; D004958:Estradiol / Q000494:pharmacology; D004965:Estrogen Antagonists / Q000494:pharmacology; D004967:Estrogens / Q000502:physiology*; D005260:Female; D005947:Glucose / Q000378:metabolism*; D009929:Organ Size / Q000187:drug effects; D010053:Ovary / Q000033:anatomy & histology; D051381:Rats; D011919:Rats, Inbred Strains; D013739:Testosterone / Q000494:pharmacology; D014599:Uterus / Q000187:drug effects / Q000378:metabolism*
chemical_list: D000728:Androgens; D004965:Estrogen Antagonists; D004967:Estrogens; D013739:Testosterone; D017373:Cyproterone Acetate; D004958:Estradiol; D003534:Cyproterone; D005947:Glucose