Pathophysiology of serous otitis media.
The likelihood that a single mechanism of immune injury is the sole cause of serous otitis media is quite small. Solid evidence is presented to suggest that type I and III immune injuries are very important, and type IV may also play a role in causing and potentiating serous otitis media in man.
D000936:Antigen-Antibody Complex / Q000276:immunology; D001327:Autoimmune Diseases / Q000276:immunology; D001402:B-Lymphocytes / Q000276:immunology; D003167:Complement Activation; D003165:Complement System Proteins / Q000276:immunology; D003602:Cytotoxicity, Immunologic; D006801:Humans; D006968:Hypersensitivity, Delayed / Q000276:immunology; D006969:Hypersensitivity, Immediate / Q000276:immunology; D007105:Immune Complex Diseases / Q000276:immunology; D007073:Immunoglobulin E / Q000276:immunology; D008247:Lysosomes / Q000201:enzymology; D010034:Otitis Media with Effusion / Q000209:etiology / Q000276:immunology*; D013601:T-Lymphocytes / Q000145:classification / Q000276:immunology
D000936:Antigen-Antibody Complex; D007073:Immunoglobulin E; D003165:Complement System Proteins