[Possible mechanisms of disorders of GTPase activity with substitutions at the 12th and 61st codons of the p21 H-ras protein].
D000595:Amino Acid Sequence; D000818:Animals; D003062:Codon / Q000235:genetics / Q000378:metabolism*; D020558:GTP Phosphohydrolases / Q000235:genetics / Q000378:metabolism*; D006801:Humans; D010744:Phosphoric Monoester Hydrolases / Q000378:metabolism*; D011485:Protein Binding; D011487:Protein Conformation; D011518:Proto-Oncogene Proteins / Q000235:genetics / Q000378:metabolism*; D016283:Proto-Oncogene Proteins p21(ras); D012333:RNA, Messenger / Q000378:metabolism*; D013329:Structure-Activity Relationship; D014170:Transformation, Genetic
D003062:Codon; D011518:Proto-Oncogene Proteins; D012333:RNA, Messenger; D010744:Phosphoric Monoester Hydrolases; D020558:GTP Phosphohydrolases; C501009:HRAS protein, human; D016283:Proto-Oncogene Proteins p21(ras)
Vozmozhnye mekhanizmy narusheniĭ GTF-aznoĭ aktivnosti pri zamenakh v 12-m i 61-m polozheniiakh belka p21 H-ras.