Evidence for a glucose effect on N-acetylglucosamine catabolism in Candida albicans.
Two strains of Candida albicans were examined for a glucose effect on the catabolism of N-acetylglucosamine. It was shown that the induction of N-acetylglucosamine uptake capacity was almost completely blocked by glucose at 0.5% (w/v), whereas that of N-acetylglucosamine kinase was partially repressed.
D000117:Acetylglucosamine / Q000378:metabolism*; D001692:Biological Transport / Q000187:drug effects; D002176:Candida albicans / Q000187:drug effects / Q000378:metabolism*; D005944:Glucosamine / Q000031:analogs & derivatives*; D005947:Glucose / Q000494:pharmacology*; D010770:Phosphotransferases / Q000378:metabolism; D017853:Phosphotransferases (Alcohol Group Acceptor); D013045:Species Specificity
D010770:Phosphotransferases; D017853:Phosphotransferases (Alcohol Group Acceptor); C018716:N-acetylglucosamine kinase; D005947:Glucose; D005944:Glucosamine; D000117:Acetylglucosamine