[A model for long-term changes in synaptic efficacy based on the interaction of the acetylcholine receptor with the 43,000 dalton sub-synaptic protein].
A model which accounts for changes in synaptic efficacy resistant to molecular turnover is presented. This model is based upon the hypothesis of a functional coupling between neurotransmitter receptor molecules by the subsynaptic protein of 43,000 daltons.
D000818:Animals; D007700:Kinetics; D008565:Membrane Proteins / Q000502:physiology*; D008959:Models, Neurological; D008970:Molecular Weight; D009419:Nerve Tissue Proteins / Q000502:physiology*; D011950:Receptors, Cholinergic / Q000502:physiology*; D013569:Synapses / Q000502:physiology*; D013570:Synaptic Membranes / Q000502:physiology; D013997:Time Factors
D008565:Membrane Proteins; D009419:Nerve Tissue Proteins; D011950:Receptors, Cholinergic
Un modèle de changement d'efficacité synaptique à long terme fondé sur l'interaction du récepteur de l'acétylcholine avec la protéine sous-synaptique de 43,000 daltons.