Participation of autonomic nerve in tonsillar focal infection.
Participation of the autonomic nerve in tonsillar focal infection was investigated by measuring neurotransmitters, receptors and microvibration. In focal infection patients, the volume of norepinephrine in the tonsil increased significantly and the number of a-adrenergic receptors decreased. These findings suggest that the focally infected tonsil exhibits a high degree of sympathetic nerve activity. The ratio of N-type in microvibration decreased systematically, which indicates some imbalance or immaturity of the autonomic nervous system in focal infection patients. Based on this data, the production mechanism of tonsillar focal infection was speculated from the point of view of the autonomic nerve.
Yamashita
T
T
Department of Otolaryngology, Kansai Medical University, Osaka, Japan.
Kozuki
K
K
Kubo
N
N
Ishibe
T
T
Kumazawa
H
H
Otani
K
K
Kumazawa
T
T
D001341:Autonomic Nervous System / Q000503:physiopathology*; D002395:Catecholamines / Q000378:metabolism; D005490:Focal Infection / Q000150:complications / Q000378:metabolism / Q000503:physiopathology*; D005533:Foot Dermatoses / Q000209:etiology / Q000378:metabolism / Q000503:physiopathology; D006229:Hand Dermatoses / Q000209:etiology / Q000378:metabolism / Q000503:physiopathology; D006801:Humans; D006984:Hypertrophy; D007674:Kidney Diseases / Q000150:complications / Q000378:metabolism; D008206:Lymphatic Diseases / Q000150:complications / Q000503:physiopathology; D014066:Palatine Tonsil / Q000294:innervation / Q000378:metabolism / Q000473:pathology; D011941:Receptors, Adrenergic / Q000378:metabolism; D014202:Tremor / Q000209:etiology / Q000503:physiopathology
D002395:Catecholamines; D011941:Receptors, Adrenergic