[Various pathogenetic mechanisms of the progressive course of chronic circulatory insufficiency in patients with ischemic heart disease].
Markedly increased antidiuretic activity, depressed epiphyseal activity and low blood serotonin, and increased sympathoadrenal activity, largely due to mediator effects, were demonstrated in 201 coronary patients with and without essential hypertension in the presence of central hemodynamic disorders. Correlations were established between impaired activities of various regulatory systems as well as between those and some hemodynamic disorders.
Rachinskiĭ
I D
ID
D000311:Adrenal Glands / Q000503:physiopathology; D000368:Aged; D002311:Cardiomyopathy, Dilated / Q000209:etiology*; D002908:Chronic Disease; D003327:Coronary Disease / Q000503:physiopathology*; D006439:Hemodynamics; D006801:Humans; D007030:Hypothalamo-Hypophyseal System / Q000503:physiopathology; D008875:Middle Aged; D013564:Sympathetic Nervous System / Q000503:physiopathology
Nekotorye patogeneticheskie mekhanizmy progressiruiushchego techeniia khronicheskoĭ nedostatochnosti krovoobrashcheniia u bol'nykh ishemicheskoĭ bolezn'iu serdtsa.