title: The beta amyloid protein (AD-AP) cDNA hybridizes in normal and Alzheimer individuals near the interface of 21q21 and q22.1.
abstract: The beta amyloid protein gene (AD-AP) has been assigned by in situ hybridization to the vicinity of the 21q21-q22.1 interface, in a normal individual as well as in a patient with Alzheimer disease.
authors.affiliation: U 173 INSERM, UA 119 CNRS, Hôpital Necker-Enfants-Malades, Paris, France.
authors.forename: V
authors.initials: V
authors.lastname: Blanquet
authors.forename: D
authors.initials: D
authors.lastname: Goldgaber
authors.forename: C
authors.initials: C
authors.lastname: Turleau
authors.forename: N
authors.initials: N
authors.lastname: Créau-Goldberg
authors.forename: J
authors.initials: J
authors.lastname: Delabar
authors.forename: P M
authors.initials: PM
authors.lastname: Sinet
authors.forename: M
authors.initials: M
authors.lastname: Roudier
authors.forename: J
authors.initials: J
authors.lastname: de Grouchy
mesh_terms: D000368:Aged; D000369:Aged, 80 and over; D000544:Alzheimer Disease / Q000235:genetics*; D000682:Amyloid / Q000235:genetics*; D016229:Amyloid beta-Peptides; D002874:Chromosome Mapping; D002891:Chromosomes, Human, Pair 21; D004247:DNA / Q000378:metabolism*; D005260:Female; D005796:Genes; D006801:Humans; D009693:Nucleic Acid Hybridization; D012016:Reference Values
chemical_list: D000682:Amyloid; D016229:Amyloid beta-Peptides; D004247:DNA