Gastric mucosal calmodulin changes in different experimental ulcer models of rat.
It seems that the mucosal CaM content does not play a significant role in the pathomechanism or the regeneration of (experimental) gastric ulcers of rat.
Balint
G A
GA
First Department of Medicine, University Medical School, Szeged, Hungary.
Varró
V
V
D000818:Animals; D002147:Calmodulin / Q000378:metabolism*; D011464:Epoprostenol / Q000378:metabolism; D005260:Female; D005748:Gastric Fundus / Q000378:metabolism; D005753:Gastric Mucosa / Q000378:metabolism*; D007213:Indomethacin; D007700:Kinetics; D011706:Pyloric Antrum / Q000378:metabolism; D051381:Rats; D011919:Rats, Inbred Strains; D013276:Stomach Ulcer / Q000139:chemically induced / Q000209:etiology / Q000378:metabolism*; D013312:Stress, Physiological / Q000503:physiopathology
D002147:Calmodulin; D011464:Epoprostenol; D007213:Indomethacin