title: Do local hemodynamic alterations mediate injury in IgA nephropathy?
authors.affiliation: Renal Unit, Royal Adelaide Hospital, Australia.
authors.forename: A J
authors.initials: AJ
authors.lastname: Woodroffe
authors.forename: K M
authors.initials: KM
authors.lastname: Bannister
authors.forename: M S
authors.initials: MS
authors.lastname: Bellon
authors.forename: G M
authors.initials: GM
authors.lastname: Hale
authors.forename: A R
authors.initials: AR
authors.lastname: Clarkson
mesh_terms: D000804:Angiotensin II / Q000032:analysis; D002216:Captopril / Q000494:pharmacology / Q000627:therapeutic use*; D002986:Clinical Trials as Topic; D005919:Glomerular Filtration Rate; D005922:Glomerulonephritis, IGA / Q000188:drug therapy / Q000503:physiopathology*; D006439:Hemodynamics / Q000187:drug effects; D006801:Humans; D007668:Kidney / Q000032:analysis; D007678:Kidney Glomerulus / Q000187:drug effects / Q000503:physiopathology*; D011897:Random Allocation; D011945:Receptors, Angiotensin / Q000032:analysis
chemical_list: D011945:Receptors, Angiotensin; D000804:Angiotensin II; D002216:Captopril