Non-psychogenic primary polydipsia in autoimmune chronic active hepatitis with severe hyperglobulinaemia.
The association of hyperglobulinaemia with renal tubular acidosis and nephrogenic diabetes insipidus is well established. A patient with marked hyperglobulinaemia due to autoimmune chronic active hepatitis is described who presented with severe polydipsia and polyuria but had entirely normal renal tubular function indicating a primary thirst disorder.
Tobin
M V
MV
Gastroenterology Unit, Royal Liverpool Hospital.
Morris
A I
AI
D000328:Adult; D001327:Autoimmune Diseases / Q000150:complications; D005260:Female; D006521:Hepatitis, Chronic / Q000150:complications / Q000523:psychology*; D006801:Humans; D011141:Polyuria / Q000209:etiology; D012712:Serum Globulins / Q000032:analysis*; D013894:Thirst / Q000502:physiology*
D012712:Serum Globulins