Hypoglycemia and hyperglycemia in tiny infants.
When the maternal supply of glucose is removed, endogenous hepatic glucose production and enteral or parenteral nutrition become the primary source of glucose supply. Because of this, the 1000-gm infant has special problems with glucose homeostasis. This article reviews these particular difficulties.
D001786:Blood Glucose / Q000378:metabolism; D001921:Brain / Q000378:metabolism; D005260:Female; D005500:Follow-Up Studies; D005934:Glucagon / Q000097:blood; D006801:Humans; D006943:Hyperglycemia / Q000097:blood* / Q000209:etiology / Q000628:therapy; D007003:Hypoglycemia / Q000097:blood* / Q000209:etiology / Q000628:therapy; D007231:Infant, Newborn; D007235:Infant, Premature, Diseases / Q000097:blood* / Q000209:etiology / Q000628:therapy; D007328:Insulin / Q000097:blood; D008099:Liver / Q000378:metabolism; D008431:Maternal-Fetal Exchange; D011247:Pregnancy
D001786:Blood Glucose; D007328:Insulin; D005934:Glucagon