Magnesium-dependent plaque formation by bacteriophage P1cinC(-) on Escherichia coli C and Shigella sonnei.
Phage P1C(-), in a state of the phage not infective to Escherichia coli K12, was able to form plaques on a wild-type strain of E. coli C and on Shigella sonnei in the presence of Mg2+. Citrobacter freundii, Enterobacter aerogenes, and a Salmonella typhimurium galE mutant were not lysed by, but were lysogenized with P1cinC(-), whereas Klebsiella pneumoniae, Proteus rettgeri, and S. typhimurium LT2 were not susceptible to either P1cinC(-) or P1cinC(+). The lipopolysaccharide structure of E. coli C and Sh. sonnei is discussed with reference to receptors for P1cinC(-) and P1cinC(+).
D001435:Bacteriophages / Q000235:genetics* / Q000254:growth & development; D004926:Escherichia coli / Q000235:genetics; D005814:Genes, Viral; D008242:Lysogeny; D008274:Magnesium / Q000502:physiology; D011991:Receptors, Virus / Q000502:physiology; D011995:Recombination, Genetic; D012764:Shigella sonnei / Q000235:genetics; D010948:Viral Plaque Assay; D014779:Virus Replication
D011991:Receptors, Virus; D008274:Magnesium