[Calcium excretion in patients with calcium oxalate calculi in acid administration].
Acidosis induced increase in renal calcium excretion was produced by means of ammonium chloride load in 12 control persons and in 76 patients with recurrent oxalate lithiasis. This increase is more marked in stone patients - even in those with normocalciuria - than in control persons. The calcium excretion is especially increased in patients with renal hypercalciuria. This might result from the primary nature of the renal defect. The acid load did not prove useful in the diagnosis of hyperparathyroidism.
D000136:Acid-Base Equilibrium / Q000187:drug effects*; D000643:Ammonium Chloride; D002118:Calcium / Q000652:urine; D002129:Calcium Oxalate / Q000652:urine*; D006801:Humans; D006961:Hyperparathyroidism / Q000652:urine; D007669:Kidney Calculi / Q000175:diagnosis / Q000652:urine*; D012008:Recurrence
D000643:Ammonium Chloride; D002129:Calcium Oxalate; D002118:Calcium
Untersuchungen zur Kalziumausscheidung bei Kalziumoxalatsteinpatienten unter Säurebelastung.