[Physiopathogenic hypothesis of alcoholic pancreatitis. Supranormal ecbolic excitation of the "pancreon" secondary to loss of the negative component of pancreatic innervation].
D000437:Alcoholism / Q000150:complications*; D000818:Animals; D001646:Bile / Q000378:metabolism; D002118:Calcium / Q000378:metabolism; D002766:Cholecystokinin / Q000378:metabolism*; D004285:Dogs; D004386:Duodenum / Q000503:physiopathology; D006801:Humans; D010179:Pancreas / Q000294:innervation / Q000503:physiopathology*; D010189:Pancreatic Juice / Q000378:metabolism; D010195:Pancreatitis / Q000503:physiopathology*; D051381:Rats
D002766:Cholecystokinin; D002118:Calcium
Hipótesis fisiopatogénica de la pancreatitis alcohólica. Excitación ecbólica supranormal del "pancreon" secundaria a la pérdida del componente negativo de la inervación pancreática.