Vitamin E deficiency in rabbits receiving a high PUFA diet with and without a non-absorbable antioxidant. II. Incorporation of 14C-labelled glycine and L-leucine into liver and plasma proteins.
As determined by in vivo studies using [1-14C] L-leucine and [1-14C] glycine, vitamin E deficiency in young rabbits caused a higher turnover rate of liver proteins and of plasma albumin and globulin fractions. This effect was most clearly and consistently observed in animals fed a diet containing 10 mg of the non-absorbable polymeric antioxidant Anoxomer per g of fat in the diet.
D000818:Animals; D000975:Antioxidants / Q000008:administration & dosage*; D001797:Blood Protein Electrophoresis; D001798:Blood Proteins / Q000378:metabolism*; D005231:Fatty Acids, Unsaturated / Q000008:administration & dosage*; D005998:Glycine / Q000097:blood*; D007930:Leucine / Q000097:blood*; D008054:Lipid Peroxides / Q000097:blood; D008099:Liver / Q000378:metabolism*; D011817:Rabbits; D012709:Serum Albumin / Q000378:metabolism; D012712:Serum Globulins / Q000378:metabolism; D014811:Vitamin E Deficiency / Q000097:blood*
D000975:Antioxidants; D001798:Blood Proteins; D005231:Fatty Acids, Unsaturated; D008054:Lipid Peroxides; D012709:Serum Albumin; D012712:Serum Globulins; D007930:Leucine; D005998:Glycine