title: Mineral X disease interactions.
abstract: The ability of animals to cope with infection may be influenced by mineral nutrition, in particular Mg and P of the macroelements and Zn, Fe, Cu and Se of the trace elements. Deficiencies of Zn, Fe, Cu and Se have resulted in a lowered resistance to disease either through an impaired immune response or faulty leukocyte function. The role of Se in bactericidal action of the phagocytes is associated with oxidative functions believed important for killing phagocytized bacteria. The role of these and other essential elements in disease prevention is an important area of animal nutrition research.
mesh_terms: D000747:Anemia, Hypochromic / Q000662:veterinary; D000824:Animal Nutritional Physiological Phenomena; D000818:Animals; D003300:Copper / Q000172:deficiency / Q000494:pharmacology; D004198:Disease Susceptibility; D007121:Immunocompetence / Q000187:drug effects; D007239:Infections / Q000276:immunology / Q000378:metabolism / Q000662:veterinary*; D007501:Iron / Q000494:pharmacology; D008274:Magnesium / Q000494:pharmacology; D008275:Magnesium Deficiency / Q000662:veterinary; D008903:Minerals / Q000378:metabolism*; D010758:Phosphorus / Q000494:pharmacology; D012643:Selenium / Q000172:deficiency / Q000494:pharmacology; D014131:Trace Elements / Q000494:pharmacology; D015032:Zinc / Q000172:deficiency / Q000494:pharmacology
chemical_list: D008903:Minerals; D014131:Trace Elements; D010758:Phosphorus; D003300:Copper; D007501:Iron; D012643:Selenium; D008274:Magnesium; D015032:Zinc