Neurovascular compression syndromes.
As I have indicated in the foregoing discussion, there are several syndromes that with various degrees of proof seem to be caused by unilateral vascular compression of a cranial nerve at the brain stem. Jannetta has summarized this concept as follows: "As we age, our arteries elongate and our brains 'sag'. As a consequence of these processes, redundant arterial loops and bridging or intrinsic hindbrain veins may cause cross-compression of cranial nerve root entry zones in the cerebellopontine angle. This pulsatile compression can be seen to produce hyperactive dysfunction of the cranial nerve. Symptoms of trigeminal or glossopharyngeal neuralgia (somatic sensory), hemifacial spasm (somatic motor), tinnitus and vertigo (special sensory), and some cases of 'essential' hypertension are caused by these vessels compressing cranial nerves V, IX-X, VII, VIII, and left X and medulla oblongata. Using microsurgical techniques, the symptoms may be relieved by vascular decompression. . .".
D000055:Accessory Nerve; D003389:Cranial Nerve Diseases / Q000175:diagnosis*; D005152:Facial Muscles; D005158:Facial Paralysis / Q000209:etiology / Q000601:surgery; D005930:Glossopharyngeal Nerve; D006801:Humans; D006973:Hypertension / Q000209:etiology; D008875:Middle Aged; D009408:Nerve Compression Syndromes / Q000175:diagnosis*; D013035:Spasm / Q000209:etiology / Q000601:surgery; D013577:Syndrome; D014103:Torticollis / Q000601:surgery; D014276:Trigeminal Nerve; D014277:Trigeminal Neuralgia / Q000601:surgery; D014630:Vagus Nerve; D000160:Vestibulocochlear Nerve Diseases / Q000209:etiology