Hepatic acid hydrolases of albino rats, Mastomys natalensis and albino mice during Plasmodium berghei infection.
Changes in liver acid hydrolase activities during the infection of albino rats, Mastomys or mice with Plasmodium berghei are described. B-Glucosidase, B-galactosidase and N-acetyl-B-D-glucosaminidase exhibited widely different responses with acid phosphatase and cathepsin-B the least responsive and are likely to be causally related to immunity of animals.
D000118:Acetylglucosaminidase / Q000378:metabolism; D000135:Acid Phosphatase / Q000378:metabolism; D000818:Animals; D002401:Cathepsin B; D002403:Cathepsins / Q000378:metabolism; D005966:Glucuronidase / Q000378:metabolism; D006867:Hydrolases / Q000378:metabolism*; D008099:Liver / Q000201:enzymology*; D008247:Lysosomes / Q000201:enzymology; D008288:Malaria / Q000201:enzymology*; D051379:Mice; D009115:Muridae; D010962:Plasmodium berghei; D051381:Rats; D001616:beta-Galactosidase / Q000378:metabolism; D001617:beta-Glucosidase / Q000378:metabolism
D006867:Hydrolases; D000135:Acid Phosphatase; D001617:beta-Glucosidase; D001616:beta-Galactosidase; D005966:Glucuronidase; D000118:Acetylglucosaminidase; D002403:Cathepsins; D002401:Cathepsin B