Mechanisms of bacterial injury.
Bacterial injury in bovine pneumonia may result from bacterial release of exotoxins or from complex interactions between bacterial products such as LPS, proteases, or antigens and host responses. The latter interactions usually result in both protection and tissue damage. The balance between degree of protective functions and injurious functions will determine whether the response is primarily beneficial or damaging to the host.
D000818:Animals; D002417:Cattle; D002418:Cattle Diseases / Q000276:immunology / Q000382:microbiology* / Q000503:physiopathology; D002630:Chemotactic Factors / Q000502:physiology; D004731:Endotoxins; D005098:Exotoxins; D006460:Hemolysin Proteins / Q000502:physiology; D010447:Peptide Hydrolases / Q000502:physiology; D011014:Pneumonia / Q000276:immunology / Q000382:microbiology / Q000503:physiopathology / Q000662:veterinary*
D002630:Chemotactic Factors; D004731:Endotoxins; D005098:Exotoxins; D006460:Hemolysin Proteins; D010447:Peptide Hydrolases