[Histamine metabolism in patients with chronic gastritis].
Examination of 104 patients has demonstrated that in marked exacerbation of chronic gastritis, surface involvement of the gastric mucosa largely localized to the fundal part, high acid-peptic activity and short-term duration of the disease, absolute hyperhistaminemia are noted. Subsiding gastritis exacerbation, the presence of glands atrophy, primary involvement of the mucous membrane in the antral part of the stomach, secretory insufficiency, as well as the disease of many years are accompanied of absolute hypohistaminemia manifesting in a low blood histamine content, the activity of histaminase and histaminopexia being unchanged or elevated.
D000293:Adolescent; D000328:Adult; D000368:Aged; D002908:Chronic Disease; D005260:Female; D005744:Gastric Acid / Q000378:metabolism; D005746:Gastric Emptying; D005748:Gastric Fundus / Q000503:physiopathology; D005756:Gastritis / Q000378:metabolism* / Q000503:physiopathology; D005757:Gastritis, Atrophic / Q000503:physiopathology; D006632:Histamine / Q000378:metabolism*; D006801:Humans; D008297:Male; D008875:Middle Aged; D010434:Pepsin A / Q000378:metabolism; D011706:Pyloric Antrum / Q000503:physiopathology
D006632:Histamine; D010434:Pepsin A
Sostoianie obmena gistamina u bol'nykh khronicheskimi gastritami.