[Succinate oxidation by rat brain mitochondria in experimental convulsions].
Corasole-induced convulsive fits are accompanied by the activation of succinate oxidation in the isolated mitochondria, paralleled by the mounting effect of factors limiting succinate dehydrogenase activity. Diverse seasonal sensitivity to corasole correlates with the inhibition of succinate-dependent energy supply for the functional neuronal activity.
D000818:Animals; D001921:Brain / Q000378:metabolism*; D008297:Male; D008928:Mitochondria / Q000378:metabolism*; D010084:Oxidation-Reduction; D010433:Pentylenetetrazole / Q000633:toxicity; D051381:Rats; D011919:Rats, Inbred Strains; D012621:Seasons; D012640:Seizures / Q000139:chemically induced / Q000378:metabolism*; D013386:Succinates / Q000378:metabolism*
D013386:Succinates; D010433:Pentylenetetrazole
Okislenie suktsinata mitokhondriiami mozga krys pri éksperimental'nom sudorozhnom pripadke.