[Structural bases for the invasion of the causative agents of experimental Pseudomonas aeruginosa sepsis into the bloodstream from the primary septic focus].
The soft tissues around the implant with a suspension of P. aeruginosa were studied by light and electron microscopy. Severe damages to structures of the histohematic barrier, phagocytes were revealed as was the bacterial invasion into thrombosed vessels of the microcirculatory bed. The mechanism of the micrometabolism as playing the key role in the bacterial penetration from the primary septic focus into the common bloodstream is discussed.
D000273:Adipose Tissue / Q000098:blood supply / Q000473:pathology; D000818:Animals; D004912:Erythrocytes / Q000473:pathology; D005490:Focal Infection / Q000382:microbiology / Q000473:pathology*; D008264:Macrophages / Q000473:pathology; D008297:Male; D008833:Microcirculation / Q000473:pathology; D008854:Microscopy, Electron; D009132:Muscles / Q000098:blood supply / Q000473:pathology; D009504:Neutrophils / Q000473:pathology; D011552:Pseudomonas Infections / Q000382:microbiology / Q000473:pathology*; D011550:Pseudomonas aeruginosa / Q000472:pathogenicity*; D051381:Rats; D018805:Sepsis / Q000382:microbiology / Q000473:pathology*
Strukturnye osnovy invazii vozbuditeleĭéksperimental'nogo sinegnoĭnogo sepsisa v krovenosnoe ruslo iz pervichnogo septicheskogo ochaga.