Sepsis and pulmonary edema.
Sepsis is the most common cause of adult respiratory distress syndrome and is associated with the highest mortality. This article describes the pathophysiology of septic pulmonary edema, which is the culmination over time of many complex responses related to sepsis.
D016718:Arachidonic Acid; D001095:Arachidonic Acids / Q000378:metabolism; D001424:Bacterial Infections / Q000150:complications / Q000503:physiopathology*; D001792:Blood Platelets / Q000502:physiology; D001794:Blood Pressure; D002199:Capillary Permeability; D004731:Endotoxins / Q000633:toxicity; D004848:Epithelium / Q000378:metabolism; D006321:Heart / Q000503:physiopathology; D006439:Hemodynamics; D006801:Humans; D000860:Hypoxia / Q000503:physiopathology; D008168:Lung / Q000503:physiopathology; D009504:Neutrophils / Q000502:physiology; D010101:Oxygen Consumption; D011650:Pulmonary Alveoli / Q000378:metabolism; D011652:Pulmonary Circulation; D011654:Pulmonary Edema / Q000209:etiology* / Q000503:physiopathology / Q000628:therapy; D011659:Pulmonary Gas Exchange; D011663:Pulmonary Surfactants / Q000502:physiology; D012128:Respiratory Distress Syndrome / Q000503:physiopathology; D012151:Resuscitation; D012772:Shock, Septic / Q000503:physiopathology; D014661:Vasoconstriction; D014882:Water-Electrolyte Balance
D001095:Arachidonic Acids; D004731:Endotoxins; D011663:Pulmonary Surfactants; D016718:Arachidonic Acid