[Thyroid dysfunction and disorders in deamination of nitrogenous compounds in experimental atherosclerosis].
A decrease in monoamine deamination with simultaneous appearance of cadaverine deaminating activity in the mitochondrial fractions of the rabbit liver, brain and heart was more noticeable under conditions of hypothyroidism combined with hypercholesterolemia developing in the presence of normal function of the thyroid. A decrease in monoamine deamination, the appearance of cadaverine deaminating activity and a significant increase in AMP deamination were observed in the mitochondrial fraction of the rabbit heart muscle under conditions of hyperthyroidism.
D000249:Adenosine Monophosphate / Q000378:metabolism; D000818:Animals; D001161:Arteriosclerosis / Q000378:metabolism*; D001596:Benzylamines / Q000378:metabolism; D002103:Cadaverine / Q000378:metabolism; D003641:Deamination; D006980:Hyperthyroidism / Q000378:metabolism*; D007037:Hypothyroidism / Q000378:metabolism*; D008297:Male; D008928:Mitochondria / Q000378:metabolism; D011817:Rabbits; D012701:Serotonin / Q000378:metabolism; D014439:Tyramine / Q000378:metabolism
D001596:Benzylamines; D012701:Serotonin; D000249:Adenosine Monophosphate; C030796:benzylamine; D002103:Cadaverine; D014439:Tyramine
Disfunktsii shchitovidnoĭ zhelezy i narusheniia dezaminirovaniia azotistykhsoedineniĭ pri ateroskleroze v éksperimente.