[Uniform theory on the pathomechanism of closed renal stalk injuries--subpelvic ureteral ruptures and kidney vascular stalk lesions].
There are no homogeneous opinions concerning the pathomechanism of closed renal stalk injuries. On the basis of a literary analysis of 168 renal vessel stalk lesions and 78 ruptures of the ureter the attempt is made to contribute to the clarification of the etiology of these injuries. Subpelvic ruptures of the ureter and renal vessel stalk lesions are regarded as a constituent of closed renal stalk injuries and traced back to a uniform pathomechanism. Deceleration traumas leading to cranial, caudal and ventral movements of the kidney are regarded as predominant cause of closed renal stalk injuries. In accidents with deceleration processes should more be thought of closed renal vessel stalk lesions and subpelvic injuries of the ureter, also when there are no blunt abdominal traumas.
D003654:Deceleration; D006801:Humans; D007682:Kidney Pelvis / Q000293:injuries*; D012077:Renal Artery / Q000293:injuries*; D012078:Renal Artery Obstruction / Q000209:etiology; D012082:Renal Veins / Q000293:injuries*; D012421:Rupture; D013927:Thrombosis / Q000209:etiology; D014513:Ureter / Q000293:injuries*; D014949:Wounds, Nonpenetrating / Q000209:etiology*
Beitrag für eine einheitliche Theorie über den Pathomechanismus geschlossener Nierenstielverletzungen--subpelvine Harnleiterrupturen und Nierengefässstielläsionen.