[Effect of stress, infarction and adaptation to brief exposure to stress on opioid peptide levels in the brain].
Content of beta-endorphine, Met- and Leu-encephalines was studied in various brain regions and in adrenal glands after long-term immobilization stress, after myocardial infarction and during prolonged gradual adaptation to short-term stressory affects. Acute stressory conditions, myocardial infarction or, especially distinct adaptation to short-term stressory affects were shown to cause an accumulation of opioid peptides in brain structures and in adrenal glands. This increase in opioid peptides accumulated as a tissue reserve appears to elevate the body resistance to subsequent injuring affects.
D000311:Adrenal Glands / Q000378:metabolism; D000818:Animals; D001921:Brain / Q000378:metabolism*; D004723:Endorphins / Q000378:metabolism*; D005790:General Adaptation Syndrome / Q000378:metabolism*; D007103:Immobilization; D008297:Male; D009203:Myocardial Infarction / Q000378:metabolism*; D011863:Radioimmunoassay; D051381:Rats; D011919:Rats, Inbred Strains; D013312:Stress, Physiological / Q000378:metabolism*; D013997:Time Factors
D004723:Endorphins
Vliianie stressa, infarkta i adaptatsii k korotkim stressornym vozdeĭstviiam na soderzhanie opioidnykh peptidov v golovnom mozge.