The morphological basis for alterations in nerve conduction in peripheral neuropathy.
D000686:Amyloidosis / Q000503:physiopathology; D000818:Animals; D001369:Axons; D001602:Beriberi / Q000503:physiopathology; D003408:Cresols; D003711:Demyelinating Diseases / Q000503:physiopathology; D003929:Diabetic Neuropathies / Q000503:physiopathology; D002549:Diffuse Cerebral Sclerosis of Schilder / Q000503:physiopathology; D004165:Diphtheria; D006801:Humans; D007538:Isoniazid; D007855:Lead Poisoning / Q000503:physiopathology; D008854:Microscopy, Electron; D009186:Myelin Sheath / Q000503:physiopathology; D009410:Nerve Degeneration; D009416:Nerve Regeneration; D009431:Neural Conduction; D009443:Neuritis / Q000503:physiopathology; D010523:Peripheral Nervous System Diseases / Q000139:chemically induced / Q000503:physiopathology*; D010710:Phosphates; D011128:Polyradiculopathy / Q000503:physiopathology; D011901:Ranvier's Nodes / Q000503:physiopathology; D012035:Refsum Disease / Q000503:physiopathology; D012583:Schwann Cells; D013792:Thalidomide
D003408:Cresols; D010710:Phosphates; D013792:Thalidomide; D007538:Isoniazid