Inhibition of Na+, K+-ATPase and digitalis action: dissociation from inotropic effects and its role in digitalis cardiotoxicity.
D000109:Acetylcholine / Q000494:pharmacology; D000251:Adenosine Triphosphatases / Q000037:antagonists & inhibitors* / Q000378:metabolism; D000450:Aldosterone / Q000494:pharmacology; D000818:Animals; D004070:Digitalis / Q000494:pharmacology*; D004077:Digoxin / Q000494:pharmacology; D004126:Dimethylformamide / Q000494:pharmacology; D004789:Enzyme Activation / Q000187:drug effects; D006207:Half-Life; D006321:Heart / Q000187:drug effects* / Q000502:physiology; D007700:Kinetics; D008861:Microsomes / Q000187:drug effects / Q000378:metabolism; D009206:Myocardium / Q000201:enzymology*; D010042:Ouabain / Q000494:pharmacology; D010477:Perfusion; D010946:Plants, Medicinal; D010947:Plants, Toxic; D011188:Potassium / Q000494:pharmacology*; D011817:Rabbits; D012964:Sodium / Q000494:pharmacology*; D013268:Stimulation, Chemical; D013327:Strophanthidin / Q000031:analogs & derivatives / Q000494:pharmacology; D013997:Time Factors; D014316:Tritium
D014316:Tritium; D000450:Aldosterone; D010042:Ouabain; D013327:Strophanthidin; D004077:Digoxin; D004126:Dimethylformamide; D012964:Sodium; D000251:Adenosine Triphosphatases; D000109:Acetylcholine; D011188:Potassium