[On the histophysiology of the rat adrenal cortex following thioacetamide (TAA) injection and ACTH stimulation].
D000135:Acid Phosphatase / Q000378:metabolism; D000311:Adrenal Glands / Q000187:drug effects; D000308:Adrenocortical Hyperfunction / Q000139:chemically induced / Q000473:pathology*; D000324:Adrenocorticotropic Hormone / Q000494:pharmacology*; D000577:Amides; D000818:Animals; D004950:Esterases / Q000378:metabolism; D006651:Histocytochemistry; D006913:Hydroxysteroid Dehydrogenases / Q000378:metabolism; D006984:Hypertrophy; D050356:Lipid Metabolism; D008297:Male; D010744:Phosphoric Monoester Hydrolases / Q000378:metabolism; D051381:Rats; D013438:Sulfhydryl Compounds
D000577:Amides; D013438:Sulfhydryl Compounds; D000324:Adrenocorticotropic Hormone; D006913:Hydroxysteroid Dehydrogenases; D004950:Esterases; D000135:Acid Phosphatase; D010744:Phosphoric Monoester Hydrolases
Uber die Histophysiologie der Nebennierenrinde bei Ratten nach Thioazetamidinjektion (TAA) und ACTH-Stimulation.