[On the mechanism of accelerated Na excretion during i.v. administration of highly hypertonic NaCl solutions in patients with essential hypertension. II. Tubular factors. Presumed functional relationships].
D000328:Adult; D000804:Angiotensin II / Q000494:pharmacology; D001795:Blood Pressure Determination; D001810:Blood Volume; D005260:Female; D006801:Humans; D006973:Hypertension / Q000503:physiopathology*; D006982:Hypertonic Solutions / Q000494:pharmacology*; D007275:Injections, Intravenous; D007671:Kidney Concentrating Ability / Q000187:drug effects; D007684:Kidney Tubules / Q000503:physiopathology*; D008297:Male; D008875:Middle Aged; D009318:Natriuresis / Q000187:drug effects*; D012965:Sodium Chloride / Q000494:pharmacology*; D014882:Water-Electrolyte Balance
D006982:Hypertonic Solutions; D000804:Angiotensin II; D012965:Sodium Chloride
Uber den Mechanismus einer beschleunigten Natriumausscheidung während intravenöser Zufuhr stark hypertonischer Kochsalzlösungen bei Patienten mit essentieller Hypertonie. II. Tubuläre Faktoren. Vermutliche funktionelle Zusammenhänge.