[Significance of the ACTH--adrenal gland system in the incidence of increased insulin sensitivity in rats with hypophyseal damage].
D000311:Adrenal Glands / Q000502:physiology; D000324:Adrenocorticotropic Hormone / Q000502:physiology; D000818:Animals; D001786:Blood Glucose / Q000032:analysis; D003348:Cortisone / Q000502:physiology; D007003:Hypoglycemia / Q000139:chemically induced; D007030:Hypothalamo-Hypophyseal System / Q000502:physiology*; D007328:Insulin / Q000494:pharmacology / Q000502:physiology*; D008297:Male; D010902:Pituitary Gland / Q000293:injuries* / Q000502:physiology; D010913:Pituitary-Adrenal System / Q000187:drug effects; D051381:Rats; D011919:Rats, Inbred Strains; D013997:Time Factors
D001786:Blood Glucose; D007328:Insulin; D000324:Adrenocorticotropic Hormone; D003348:Cortisone
Die Bedeutung des ACTH--Nebennierenrindensystems bei der Entstehung gesteigerter Insulinempfindlichkeit Hypophysenstiel-lädierter Ratten.