[Pathogenesis of ascites formation in liver cirrhosis (author's transl)].
D000450:Aldosterone / Q000097:blood; D000804:Angiotensin II / Q000097:blood; D001201:Ascites / Q000097:blood / Q000209:etiology* / Q000503:physiopathology; D002199:Capillary Permeability; D006439:Hemodynamics; D006801:Humans; D006929:Hyperaldosteronism / Q000150:complications; D006975:Hypertension, Portal / Q000150:complications / Q000209:etiology; D008103:Liver Cirrhosis / Q000097:blood / Q000150:complications* / Q000503:physiopathology; D008196:Lymph / Q000502:physiology; D008208:Lymphatic System / Q000503:physiopathology; D009997:Osmotic Pressure; D012083:Renin / Q000097:blood
D000804:Angiotensin II; D000450:Aldosterone; D012083:Renin
Pathogenese des Aszites bei der Leberzirrhose