A possible mechanism for the stimulation of Krebs cycle activity by ACTH in isolated rat adrenal cells.
D000311:Adrenal Glands / Q000187:drug effects / Q000378:metabolism*; D000324:Adrenocorticotropic Hormone / Q000494:pharmacology*; D000616:Aminoglutethimide / Q000494:pharmacology; D000818:Animals; D003994:Bucladesine / Q000494:pharmacology; D002245:Carbon Dioxide; D002250:Carbon Radioisotopes; D002952:Citric Acid Cycle / Q000187:drug effects*; D003345:Corticosterone / Q000096:biosynthesis / Q000378:metabolism / Q000494:pharmacology; D066298:In Vitro Techniques; D007700:Kinetics; D003012:Microbial Collagenase; D011284:Pregnenolone / Q000378:metabolism / Q000494:pharmacology; D011374:Progesterone / Q000378:metabolism / Q000494:pharmacology; D011773:Pyruvates / Q000378:metabolism; D051381:Rats; D014357:Trypsin
D002250:Carbon Radioisotopes; D011773:Pyruvates; D000616:Aminoglutethimide; D002245:Carbon Dioxide; D011374:Progesterone; D003994:Bucladesine; D011284:Pregnenolone; D000324:Adrenocorticotropic Hormone; D014357:Trypsin; D003012:Microbial Collagenase; D003345:Corticosterone