[Pathogenesis of the septic-toxic shock].
D000818:Animals; D001329:Autolysis; D002199:Capillary Permeability; D002302:Cardiac Output; D002403:Cathepsins / Q000097:blood; D004211:Disseminated Intravascular Coagulation / Q000209:etiology; D005966:Glucuronidase / Q000097:blood; D006801:Humans; D007022:Hypotension / Q000209:etiology; D008247:Lysosomes / Q000378:metabolism; D010101:Oxygen Consumption; D010744:Phosphoric Monoester Hydrolases / Q000097:blood; D011239:Prednisolone / Q000627:therapeutic use; D011379:Prognosis; D018805:Sepsis / Q000150:complications; D012772:Shock, Septic / Q000097:blood / Q000150:complications / Q000201:enzymology / Q000209:etiology* / Q000378:metabolism / Q000628:therapy; D012790:Shwartzman Phenomenon / Q000503:physiopathology; D009626:Terminology as Topic; D013997:Time Factors; D014884:Waterhouse-Friderichsen Syndrome / Q000503:physiopathology
D011239:Prednisolone; D010744:Phosphoric Monoester Hydrolases; D005966:Glucuronidase; D002403:Cathepsins
Zur Pathogenese des septisch-toxischen Schocks.