Mechanism of hypertriglyceridaemia associated with contraceptive steroids.
D000293:Adolescent; D000328:Adult; D001835:Body Weight; D002247:Carbon Isotopes; D003276:Contraceptives, Oral / Q000494:pharmacology*; D004997:Ethinyl Estradiol / Q000494:pharmacology; D005040:Ethynodiol Diacetate / Q000494:pharmacology; D005230:Fatty Acids, Nonesterified / Q000097:blood* / Q000378:metabolism; D005260:Female; D006493:Heparin / Q000494:pharmacology; D006801:Humans; D007330:Insulin Antibodies; D007700:Kinetics; D008079:Lipoproteins, VLDL / Q000378:metabolism / Q000494:pharmacology; D008535:Megestrol / Q000494:pharmacology; D008656:Mestranol / Q000494:pharmacology; D009640:Norethindrone / Q000494:pharmacology; D010169:Palmitic Acids / Q000378:metabolism; D011374:Progesterone / Q000494:pharmacology; D013997:Time Factors; D014280:Triglycerides / Q000097:blood* / Q000378:metabolism
D002247:Carbon Isotopes; D003276:Contraceptives, Oral; D005230:Fatty Acids, Nonesterified; D007330:Insulin Antibodies; D008079:Lipoproteins, VLDL; D010169:Palmitic Acids; D014280:Triglycerides; D004997:Ethinyl Estradiol; D011374:Progesterone; D005040:Ethynodiol Diacetate; D006493:Heparin; D008656:Mestranol; D008535:Megestrol; D009640:Norethindrone