[Possible role of slow sodium-calcium channels in the mechanism of changes in the electrical and mechanical activity of guinea pig myocardial cells under local anaphylaxis (isoptin effects)].
D000707:Anaphylaxis; D000818:Animals; D001693:Biological Transport, Active / Q000187:drug effects; D002118:Calcium / Q000378:metabolism*; D004594:Electrophysiology; D006168:Guinea Pigs; D006321:Heart / Q000503:physiopathology*; D006325:Heart Atria; D006327:Heart Block / Q000139:chemically induced / Q000503:physiopathology; D006339:Heart Rate / Q000187:drug effects; D006632:Histamine / Q000494:pharmacology; D007114:Immunization; D066298:In Vitro Techniques; D008839:Microelectrodes; D009206:Myocardium / Q000166:cytology* / Q000378:metabolism; D010047:Ovalbumin / Q000276:immunology; D010477:Perfusion; D012964:Sodium / Q000378:metabolism*; D014700:Verapamil / Q000031:analogs & derivatives* / Q000494:pharmacology
D006632:Histamine; D010047:Ovalbumin; D012964:Sodium; D014700:Verapamil; D002118:Calcium
O vozmozhnoĭ roli medlennykh natriĭ-kal'tsievykh kanalov v mekhanizme izmeneniĭ élektricheskoĭ i mekhanicheskoĭ aktivnosti kletok serdtsa morskoĭ svinki pri mestnoĭ anafilaksii (éffeky izoptina)