Myocardial fiber necrosis due to intracellular Ca overload-a new principle in cardiac pathophysiology.
D000643:Ammonium Chloride / Q000494:pharmacology; D000818:Animals; D001692:Biological Transport / Q000187:drug effects; D002116:Calcitonin / Q000494:pharmacology; D002118:Calcium / Q000378:metabolism*; D009202:Cardiomyopathies / Q000139:chemically induced / Q000378:metabolism; D004097:Dihydrotachysterol / Q000494:pharmacology; D004305:Dose-Response Relationship, Drug; D004357:Drug Synergism; D006168:Guinea Pigs; D006321:Heart / Q000187:drug effects*; D006863:Hydrogen-Ion Concentration; D007545:Isoproterenol / Q000037:antagonists & inhibitors / Q000494:pharmacology*; D008274:Magnesium / Q000494:pharmacology; D009206:Myocardium / Q000378:metabolism; D009336:Necrosis; D010710:Phosphates / Q000378:metabolism; D011188:Potassium / Q000494:pharmacology; D011299:Prenylamine / Q000494:pharmacology; D051381:Rats; D014700:Verapamil / Q000494:pharmacology
D010710:Phosphates; D000643:Ammonium Chloride; D002116:Calcitonin; D014700:Verapamil; D008274:Magnesium; D011299:Prenylamine; D007545:Isoproterenol; D004097:Dihydrotachysterol; D011188:Potassium; D002118:Calcium