Novel mutation to dominant fucose resistance in the L-arabinose operon of Escherichia coli.
We isolated an unusual mutant in which the arabinose operon carried an araC lesion that led to a dominant fucose resistance while retaining recessive constitutivity. All previously characterized C(c) mutations have been recessive for both fucose resistance and constitutivity.
D001089:Arabinose / Q000378:metabolism*; D002238:Carbohydrate Epimerases / Q000378:metabolism; D002247:Carbon Isotopes; D004352:Drug Resistance, Microbial; D004926:Escherichia coli / Q000187:drug effects / Q000201:enzymology / Q000254:growth & development / Q000302:isolation & purification / Q000378:metabolism*; D005643:Fucose / Q000494:pharmacology*; D005799:Genes, Dominant; D009153:Mutagens; D009154:Mutation; D009604:Nitrosoguanidines; D009876:Operon; D013237:Stereoisomerism
D002247:Carbon Isotopes; D009153:Mutagens; D009604:Nitrosoguanidines; D005643:Fucose; D001089:Arabinose; D002238:Carbohydrate Epimerases