The role of acid in the pathogenesis of aspirin-induced gastrointestinal erosions and hemorrhage.
D000818:Animals; D001205:Ascorbic Acid / Q000378:metabolism; D001241:Aspirin / Q000008:administration & dosage / Q000009:adverse effects* / Q000378:metabolism / Q000627:therapeutic use; D001778:Blood Coagulation Disorders / Q000150:complications; D002021:Buffers; D004285:Dogs; D004304:Dosage Forms; D004357:Drug Synergism; D000431:Ethanol / Q000494:pharmacology; D005750:Gastric Juice / Q000378:metabolism*; D005753:Gastric Mucosa / Q000187:drug effects* / Q000378:metabolism; D006168:Guinea Pigs; D006801:Humans; D006863:Hydrogen-Ion Concentration; D007408:Intestinal Absorption; D007962:Leukocytes / Q000378:metabolism; D009780:Occult Blood; D010438:Peptic Ulcer Hemorrhage / Q000139:chemically induced*; D010539:Permeability; D011817:Rabbits; D051381:Rats; D013276:Stomach Ulcer / Q000139:chemically induced* / Q000517:prevention & control
D002021:Buffers; D004304:Dosage Forms; D000431:Ethanol; D001205:Ascorbic Acid; D001241:Aspirin