Acidosis induced by catecholamines and reduction of cardiovascular responses to catecholamines in acidosis.
D000138:Acidosis / Q000139:chemically induced / Q000503:physiopathology*; D000818:Animals; D001769:Blood; D001794:Blood Pressure / Q000187:drug effects*; D002245:Carbon Dioxide / Q000097:blood; D002395:Catecholamines / Q000494:pharmacology*; D004357:Drug Synergism; D004837:Epinephrine / Q000494:pharmacology; D005260:Female; D006339:Heart Rate / Q000187:drug effects*; D006851:Hydrochloric Acid; D006863:Hydrogen-Ion Concentration; D007545:Isoproterenol / Q000037:antagonists & inhibitors / Q000494:pharmacology; D008297:Male; D009638:Norepinephrine / Q000037:antagonists & inhibitors / Q000494:pharmacology; D010313:Partial Pressure; D010643:Phenoxybenzamine / Q000494:pharmacology; D011433:Propranolol / Q000494:pharmacology; D051381:Rats; D014325:Tromethamine / Q000494:pharmacology
D002395:Catecholamines; D014325:Tromethamine; D010643:Phenoxybenzamine; D002245:Carbon Dioxide; D011433:Propranolol; D007545:Isoproterenol; D006851:Hydrochloric Acid; D009638:Norepinephrine; D004837:Epinephrine