Role of the acidosis in the development of increased pulmonary vascular resistance and shock lung in experimental hemorrhagic shock.
D000138:Acidosis / Q000150:complications / Q000503:physiopathology*; D000818:Animals; D002245:Carbon Dioxide / Q000097:blood; D004195:Disease Models, Animal; D004285:Dogs; D005260:Female; D006439:Hemodynamics; D006863:Hydrogen-Ion Concentration; D008168:Lung; D008171:Lung Diseases / Q000209:etiology*; D008297:Male; D009929:Organ Size; D010100:Oxygen / Q000097:blood; D011650:Pulmonary Alveoli; D011651:Pulmonary Artery; D011652:Pulmonary Circulation; D012771:Shock, Hemorrhagic / Q000150:complications / Q000503:physiopathology*; D014655:Vascular Resistance
D002245:Carbon Dioxide; D010100:Oxygen