Calcium-induced ciliary reversal in the extracted models of "Pawn", a behavioral mutant of Paramecium.
"Pawn," a genic mutant of Paramecium aurelia, cannot swim backward as the wild type can upon proper stimulation. In contrast, after membrane disruption by Triton X-100, the adenosine triphosphate-magnesium reactivated models of Pawns swim backward in the presence of calcium as wild-type models do. Thus, the mutant phenotype is due to an impairment in the membrane and not in the calcium-sensitive motile system.
D000255:Adenosine Triphosphate / Q000378:metabolism; D000818:Animals; D002118:Calcium / Q000378:metabolism / Q000494:pharmacology*; D002463:Cell Membrane Permeability; D002923:Cilia / Q000187:drug effects / Q000502:physiology*; D008124:Locomotion / Q000187:drug effects; D008274:Magnesium / Q000378:metabolism; D009154:Mutation; D010247:Paramecium / Q000166:cytology / Q000187:drug effects / Q000378:metabolism / Q000502:physiology*; D013501:Surface-Active Agents / Q000494:pharmacology
D013501:Surface-Active Agents; D000255:Adenosine Triphosphate; D008274:Magnesium; D002118:Calcium