On the molecular basis of pyruvate kinase deficiency. I. Primary defect or conseqence of increased glutathione disulfide concentration.
D000328:Adult; D000494:Allosteric Regulation; D004220:Disulfides / Q000494:pharmacology*; D004355:Drug Stability; D004912:Erythrocytes / Q000187:drug effects / Q000201:enzymology*; D005636:Fructosephosphates / Q000494:pharmacology; D005978:Glutathione / Q000494:pharmacology*; D006598:Hexosediphosphates / Q000494:pharmacology; D006358:Hot Temperature; D006801:Humans; D007700:Kinetics; D010084:Oxidation-Reduction; D011770:Pyruvate Kinase / Q000097:blood*; D013056:Spectrophotometry, Ultraviolet; D013997:Time Factors
D004220:Disulfides; D005636:Fructosephosphates; D006598:Hexosediphosphates; D011770:Pyruvate Kinase; D005978:Glutathione