Shock following acute myocardial infarction. Current understanding of hemodynamic mechanisms.
D000138:Acidosis / Q000209:etiology; D000450:Aldosterone / Q000378:metabolism; D000818:Animals; D001145:Arrhythmias, Cardiac; D001158:Arteries / Q000503:physiopathology; D001783:Blood Flow Velocity; D001794:Blood Pressure; D001810:Blood Volume; D002302:Cardiac Output; D003327:Coronary Disease / Q000503:physiopathology; D003331:Coronary Vessels / Q000503:physiopathology; D048909:Diabetes Complications; D006331:Heart Diseases / Q000503:physiopathology; D006333:Heart Failure; D006352:Heart Ventricles / Q000503:physiopathology; D006439:Hemodynamics; D006801:Humans; D006973:Hypertension / Q000150:complications; D009203:Myocardial Infarction / Q000150:complications* / Q000503:physiopathology; D010101:Oxygen Consumption; D011086:Polycythemia / Q000150:complications; D011652:Pulmonary Circulation; D012018:Reflex / Q000502:physiology; D012769:Shock / Q000453:epidemiology / Q000209:etiology* / Q000503:physiopathology; D014655:Vascular Resistance; D014680:Veins / Q000503:physiopathology
D000450:Aldosterone