[Importance of hyperammoniemia of the renal origin in the pathogenesis of hepatic coma released in cirrhotic patients by diuretics generating hypokaliemia].
D000086:Acetazolamide / Q000009:adverse effects*; D000136:Acid-Base Equilibrium; D000471:Alkalosis / Q000139:chemically induced; D000641:Ammonia / Q000097:blood* / Q000378:metabolism / Q000652:urine; D001201:Ascites / Q000188:drug therapy; D002712:Chlorides / Q000097:blood; D002752:Chlorthalidone / Q000009:adverse effects*; D004487:Edema / Q000188:drug therapy; D004976:Ethacrynic Acid / Q000009:adverse effects*; D005665:Furosemide / Q000009:adverse effects*; D006501:Hepatic Encephalopathy / Q000139:chemically induced* / Q000503:physiopathology; D006801:Humans; D006863:Hydrogen-Ion Concentration; D007008:Hypokalemia / Q000139:chemically induced; D007668:Kidney / Q000503:physiopathology; D008103:Liver Cirrhosis / Q000097:blood / Q000188:drug therapy* / Q000503:physiopathology; D012964:Sodium / Q000097:blood; D014882:Water-Electrolyte Balance / Q000187:drug effects
D002712:Chlorides; D000641:Ammonia; D005665:Furosemide; D012964:Sodium; D004976:Ethacrynic Acid; D000086:Acetazolamide; D002752:Chlorthalidone
Importance de l'hyperammoniémie d'origine rénale dans la pathogénie des comas hépatiques déclenchés chez les cirrhotiques par les diurétiques générateurs d'hypokaliémie.