Spontaneous hyperglycemia and-or obesity in laboratory rodents: an example of the possible usefulness of animal disease models with both genetic and environmental components.
D000138:Acidosis / Q000503:physiopathology; D000273:Adipose Tissue / Q000503:physiopathology; D000818:Animals; D000830:Animals, Laboratory; D001786:Blood Glucose / Q000032:analysis; D003920:Diabetes Mellitus / Q000235:genetics / Q000503:physiopathology; D004195:Disease Models, Animal; D005247:Feeding Behavior; D006003:Glycogen / Q000032:analysis; D006168:Guinea Pigs; D000882:Haplorhini; D006943:Hyperglycemia / Q000235:genetics* / Q000503:physiopathology; D006946:Hyperinsulinism / Q000503:physiopathology; D007328:Insulin / Q000096:biosynthesis / Q000097:blood / Q000378:metabolism / Q000494:pharmacology; D007333:Insulin Resistance; D000078790:Insulin Secretion; D007515:Islets of Langerhans / Q000473:pathology; D007657:Ketone Bodies / Q000096:biosynthesis; D051379:Mice; D009132:Muscles / Q000032:analysis; D009765:Obesity; D010179:Pancreas / Q000032:analysis; D011236:Prediabetic State / Q000235:genetics / Q000503:physiopathology; D051381:Rats
D001786:Blood Glucose; D007328:Insulin; D007657:Ketone Bodies; D006003:Glycogen