Role of the redox state in ethanol-induced suppression of citrate-cycle flux in the perfused liver of normal, hyper- and hypothyroid rats.
D000079:Acetaldehyde / Q000494:pharmacology; D000085:Acetates; D000818:Animals; D002208:Caproates; D002245:Carbon Dioxide / Q000378:metabolism; D002247:Carbon Isotopes; D002952:Citric Acid Cycle / Q000187:drug effects*; D000431:Ethanol / Q000378:metabolism / Q000494:pharmacology*; D006980:Hyperthyroidism / Q000139:chemically induced / Q000378:metabolism*; D007037:Hypothyroidism / Q000139:chemically induced / Q000378:metabolism*; D066298:In Vitro Techniques; D007773:Lactates; D008099:Liver / Q000187:drug effects / Q000378:metabolism*; D008297:Male; D010088:Oxidoreductases / Q000378:metabolism; D010477:Perfusion; D011441:Propylthiouracil; D011720:Pyrazoles / Q000494:pharmacology; D011773:Pyruvates; D051381:Rats; D014284:Triiodothyronine
D000085:Acetates; D002208:Caproates; D002247:Carbon Isotopes; D007773:Lactates; D011720:Pyrazoles; D011773:Pyruvates; D014284:Triiodothyronine; D002245:Carbon Dioxide; D000431:Ethanol; D011441:Propylthiouracil; D010088:Oxidoreductases; D000079:Acetaldehyde