[Gradual lesions of the wake system as a pathophysiological mechanism of the mode of action of cerebral noxae].
D000328:Adult; D001924:Brain Concussion / Q000503:physiopathology; D001927:Brain Diseases / Q000503:physiopathology*; D001933:Brain Stem / Q000502:physiology / Q000503:physiopathology*; D002551:Cerebral Ventricle Neoplasms / Q000503:physiopathology; D002648:Child; D003344:Cortical Synchronization; D004569:Electroencephalography; D004660:Encephalitis / Q000503:physiopathology; D004806:Ependymoma / Q000503:physiopathology; D004827:Epilepsy / Q000503:physiopathology; D004833:Epilepsy, Temporal Lobe / Q000503:physiopathology; D006221:Halothane; D006801:Humans; D008875:Middle Aged; D008881:Migraine Disorders / Q000503:physiopathology; D009434:Neural Pathways / Q000502:physiology; D012154:Reticular Formation / Q000503:physiopathology; D012890:Sleep; D013788:Thalamus / Q000502:physiology; D014851:Wakefulness
D006221:Halothane
Die gestufte Schädigung des Wachsystems als ein pathophysiologischer Mechanismus der Wirkungsweise zerebraler Noxen.