The role of electrolyte disturbances and extracellular alkalosis in metabolic cardiac necrosis and the preventive effect of amiloride.
D000136:Acid-Base Equilibrium; D000471:Alkalosis / Q000139:chemically induced / Q000378:metabolism; D000818:Animals; D001639:Bicarbonates / Q000097:blood; D002245:Carbon Dioxide / Q000097:blood; D002712:Chlorides / Q000097:blood; D004573:Electrolytes / Q000378:metabolism*; D005260:Female; D005438:Fludrocortisone; D006331:Heart Diseases / Q000209:etiology / Q000378:metabolism / Q000517:prevention & control*; D006863:Hydrogen-Ion Concentration; D006955:Hypernatremia / Q000139:chemically induced; D007008:Hypokalemia / Q000139:chemically induced; D008055:Lipids / Q000008:administration & dosage; D008274:Magnesium / Q000032:analysis / Q000097:blood; D009132:Muscles / Q000032:analysis / Q000378:metabolism; D009206:Myocardium / Q000032:analysis / Q000378:metabolism*; D009336:Necrosis; D010710:Phosphates / Q000494:pharmacology; D011188:Potassium / Q000032:analysis / Q000097:blood; D051381:Rats; D012964:Sodium / Q000032:analysis / Q000378:metabolism
D001639:Bicarbonates; D002712:Chlorides; D004573:Electrolytes; D008055:Lipids; D010710:Phosphates; D002245:Carbon Dioxide; D012964:Sodium; D008274:Magnesium; D011188:Potassium; D005438:Fludrocortisone