[Biochemical mechanisms of steatosis as sequelae of "nucleotide imbalance"].
D000085:Acetates / Q000378:metabolism; D000225:Adenine / Q000494:pharmacology; D000227:Adenine Nucleotides / Q000378:metabolism*; D000255:Adenosine Triphosphate / Q000032:analysis; D000818:Animals; D001377:Azaserine; D001710:Biotin / Q000032:analysis; D002247:Carbon Isotopes; D002784:Cholesterol / Q000032:analysis; D004195:Disease Models, Animal; D005001:Ethionine; D005234:Fatty Liver / Q000139:chemically induced / Q000378:metabolism*; D050356:Lipid Metabolism; D008055:Lipids / Q000032:analysis; D009711:Nucleotides / Q000378:metabolism; D009963:Orotic Acid; D010743:Phospholipids / Q000032:analysis; D011720:Pyrazoles; D011743:Pyrimidines; D051381:Rats; D014280:Triglycerides / Q000378:metabolism
D000085:Acetates; D000227:Adenine Nucleotides; D002247:Carbon Isotopes; D008055:Lipids; D009711:Nucleotides; D010743:Phospholipids; D011720:Pyrazoles; D011743:Pyrimidines; D014280:Triglycerides; D009963:Orotic Acid; D001710:Biotin; D001377:Azaserine; D000255:Adenosine Triphosphate; D002784:Cholesterol; D000225:Adenine; D005001:Ethionine
Biochemische Mechanismen der Steatose als Folge einer "Nukleotid-Gleichgewichtsstörung".