[Effect of galascorbin in respiration and oxidative phosphorylation of mitochondria of liver affected by carbon tetrachloride poisoning].
D000818:Animals; D001205:Ascorbic Acid / Q000494:pharmacology* / Q000627:therapeutic use; D002252:Carbon Tetrachloride Poisoning / Q000378:metabolism* / Q000517:prevention & control; D056486:Chemical and Drug Induced Liver Injury / Q000188:drug therapy / Q000378:metabolism*; D003879:Dermatologic Agents / Q000494:pharmacology* / Q000627:therapeutic use; D008930:Mitochondria, Liver / Q000378:metabolism*; D010085:Oxidative Phosphorylation / Q000187:drug effects; D010101:Oxygen Consumption / Q000187:drug effects; D051381:Rats; D013386:Succinates / Q000378:metabolism; D013634:Tannins / Q000494:pharmacology* / Q000627:therapeutic use
D003879:Dermatologic Agents; D013386:Succinates; D013634:Tannins; D001205:Ascorbic Acid
Vplyv halaskorbinu na dykhannia ta okysne fosforyliuvannia mitokhondriĭ pechinky shchuriv pry ïï ushkodzhenni chotyrykhlorystym vuhletsem.