The role of inactivation in the cumulative blockage of voltage-dependent sodium channels by local anesthetics and antiarrythmics.
D000779:Anesthetics, Local / Q000494:pharmacology*; D000818:Animals; D000889:Anti-Arrhythmia Agents / Q000494:pharmacology*; D002700:Chloramines / Q000494:pharmacology; D004594:Electrophysiology; D007473:Ion Channels / Q000187:drug effects / Q000502:physiology*; D008012:Lidocaine / Q000494:pharmacology; D011219:Prajmaline / Q000494:pharmacology; D011412:Propanolamines / Q000494:pharmacology; D011895:Rana ridibunda; D012964:Sodium / Q000378:metabolism*; D013748:Tetracaine / Q000494:pharmacology; D014105:Tosyl Compounds
D000779:Anesthetics, Local; D000889:Anti-Arrhythmia Agents; D002700:Chloramines; D007473:Ion Channels; D011412:Propanolamines; D014105:Tosyl Compounds; C016300:chloramine-T; D013748:Tetracaine; D011219:Prajmaline; C044763:KC 3791; D008012:Lidocaine; D012964:Sodium