title: Airway hyperreactivity. Clinical treatment with drugs.
abstract: At present there is no study indicating that any drug can decrease "primary hyperreactivity". If the frequency of asthma attacks can be lowered this seems to cause a decrease of the induced (?) hyperreactivity. This is proven for DCG and gold and may also be true for steroids and perhaps for other effective anti-asthmatic drugs. The mechanism is probably a decrease of the inflammatory reaction in the bronchial mucosa due to less frequent and less severe asthma attacks.
mesh_terms: D000109:Acetylcholine / Q000494:pharmacology; D000305:Adrenal Cortex Hormones / Q000627:therapeutic use; D000318:Adrenergic beta-Agonists / Q000627:therapeutic use; D001249:Asthma / Q000188:drug therapy*; D001985:Bronchial Provocation Tests; D004205:Cromolyn Sodium / Q000627:therapeutic use; D006046:Gold / Q000627:therapeutic use; D006632:Histamine; D006801:Humans; D007665:Ketotifen / Q000627:therapeutic use; D012130:Respiratory Hypersensitivity / Q000188:drug therapy*; D013726:Terbutaline / Q000627:therapeutic use
chemical_list: D000305:Adrenal Cortex Hormones; D000318:Adrenergic beta-Agonists; D006046:Gold; D006632:Histamine; D013726:Terbutaline; D000109:Acetylcholine; D004205:Cromolyn Sodium; D007665:Ketotifen