Airway hyperreactivity. Clinical treatment with drugs.
At present there is no study indicating that any drug can decrease "primary hyperreactivity". If the frequency of asthma attacks can be lowered this seems to cause a decrease of the induced (?) hyperreactivity. This is proven for DCG and gold and may also be true for steroids and perhaps for other effective anti-asthmatic drugs. The mechanism is probably a decrease of the inflammatory reaction in the bronchial mucosa due to less frequent and less severe asthma attacks.
D000109:Acetylcholine / Q000494:pharmacology; D000305:Adrenal Cortex Hormones / Q000627:therapeutic use; D000318:Adrenergic beta-Agonists / Q000627:therapeutic use; D001249:Asthma / Q000188:drug therapy*; D001985:Bronchial Provocation Tests; D004205:Cromolyn Sodium / Q000627:therapeutic use; D006046:Gold / Q000627:therapeutic use; D006632:Histamine; D006801:Humans; D007665:Ketotifen / Q000627:therapeutic use; D012130:Respiratory Hypersensitivity / Q000188:drug therapy*; D013726:Terbutaline / Q000627:therapeutic use
D000305:Adrenal Cortex Hormones; D000318:Adrenergic beta-Agonists; D006046:Gold; D006632:Histamine; D013726:Terbutaline; D000109:Acetylcholine; D004205:Cromolyn Sodium; D007665:Ketotifen