[Cornea and neovascularisation: a physiopathological study (author's transl)].
Analysis of clinical data and reports in the published literature are employed to develop a physiopathological explanation for corneal neovascularisation, based on a chain reaction in which inflammation and hypoxia play essential roles. The possible advantages and disadvantages that result from this neovascularisation are analysed. There is no fundamental reason for the administration of antivascular therapy alone, and treatment should be directed towards the inflammatory process, which is the cause of the neovascularisation and the opacities. By reducing or suppressing the inflammatory chain reaction it is then possible to break the vicious circle of a possible self-supporting affection.
D000305:Adrenal Cortex Hormones / Q000627:therapeutic use; D003261:Contact Lenses / Q000009:adverse effects; D003315:Cornea / Q000098:blood supply* / Q000503:physiopathology; D003318:Corneal Opacity / Q000209:etiology; D016039:Corneal Transplantation; D006801:Humans; D007634:Keratitis / Q000188:drug therapy / Q000503:physiopathology; D009389:Neovascularization, Pathologic; D010101:Oxygen Consumption; D011453:Prostaglandins / Q000502:physiology
D000305:Adrenal Cortex Hormones; D011453:Prostaglandins
Cornée et néovascularisation. Essai physiopathologique.