On the mechanism of the augmentation of the phagocytic capability of phagocytic cells by Tuftsin, substance P, neurotensin, and kentsin and the interrelationship between their receptors.
D000818:Animals; D001667:Binding, Competitive; D002118:Calcium / Q000378:metabolism; D000242:Cyclic AMP / Q000378:metabolism; D006152:Cyclic GMP / Q000378:metabolism; D004305:Dose-Response Relationship, Drug; D004912:Erythrocytes; D006801:Humans; D008264:Macrophages / Q000378:metabolism; D051379:Mice; D009496:Neurotensin / Q000494:pharmacology*; D009504:Neutrophils / Q000378:metabolism; D009842:Oligopeptides / Q000494:pharmacology*; D010586:Phagocytes / Q000378:metabolism; D010587:Phagocytosis / Q000187:drug effects*; D011956:Receptors, Cell Surface / Q000502:physiology*; D011971:Receptors, Immunologic; D013329:Structure-Activity Relationship; D013373:Substance P / Q000494:pharmacology*; D014405:Tuftsin / Q000378:metabolism / Q000494:pharmacology*; D015003:Yeasts
D009842:Oligopeptides; D011956:Receptors, Cell Surface; D011971:Receptors, Immunologic; C029695:tuftsin receptor; D013373:Substance P; D009496:Neurotensin; C017147:kentsin; D000242:Cyclic AMP; D006152:Cyclic GMP; D014405:Tuftsin; D002118:Calcium