[Interrelation of the processes of energy release and demand in the myocardium with its contractile function in acquired heart defects].
Patients with acquired heart defects show oxidative phosphorylation disorders and reduced actomyosin ATPase activity. The markedness of changes is proportionate to the severity of valvular lesion as well as heart and myocardial failure. A relationship is demonstrated between disorders of energy generation and utilization, and contractility changes in the myocardium.
D000205:Actomyosin / Q000378:metabolism; D000251:Adenosine Triphosphatases / Q000378:metabolism; D001022:Aortic Valve Insufficiency / Q000503:physiopathology*; D004734:Energy Metabolism; D006352:Heart Ventricles / Q000378:metabolism; D006801:Humans; D008929:Mitochondria, Heart / Q000378:metabolism; D008944:Mitral Valve Insufficiency / Q000503:physiopathology*; D009200:Myocardial Contraction; D009206:Myocardium / Q000378:metabolism*; D010085:Oxidative Phosphorylation
D000205:Actomyosin; D000251:Adenosine Triphosphatases
Vzaimosviaz' protsessov osvobozhdeniia i potrebleniia énergii v miokarde s ego sokratitel'noĭ funktsieĭ pri priobretennykh porokakh serdtsa.