[A contribution to the electrogenetic interpretation of left ventricular hypertrophy caused by volume overload].
The purpose of the present study is to make a contribution to the understanding of the electrogenetic interpretation of left ventricular hypertrophy caused by volume overload. Thirty-two cases of isolated aortic valve regurgitation with electrocardiographic evidence of left ventricular hypertrophy were studied by vectorcardiography in order to measure the 20 msec vector both in the spatial and horizontal plane, and by M-Mode echocardiography to obtain the interventricular septal and posterior wall thickness and the end diastolic dimension of the left ventricle. The following relations were analyzed: a) between the interventricular septal thickness and the amplitude of the 20 msec vector in the spatial and horizontal plane; b) between the end diastolic dimension of the left ventricle and the amplitude of the 20 msec vector. We also explored the possibility to distinguish by means of the 20 msec vector amplitude between patients with and without disproportionate septal thickening. We found no correlation either between interventricular septal thickness and amplitude of the 20 msec vector, or between left ventricular end diastolic dimension and amplitude of the 20 msec vector. It was not possible to distinguish by means of the amplitude of the 20 msec vector between the cases of left ventricular hypertrophy with disproportional septal thickening and those without it.
D000293:Adolescent; D000328:Adult; D000368:Aged; D002306:Cardiac Volume; D006332:Cardiomegaly / Q000175:diagnosis* / Q000473:pathology / Q000503:physiopathology; D004452:Echocardiography; D004562:Electrocardiography; D006801:Humans; D008875:Middle Aged; D014672:Vectorcardiography
Contributo all'interpretazione elettrogenetica della ipertrofia ventricolare sinistra da sovraccarico di volume.