[Etiopathogenesis of colorectal carcinomas].
There are a small group of colonic carcinomas of known etiology and large group of unknown etiology. In the latter group epidemiology, metabolic epidemiology, and animal experiments suggest a correlation between the fat content of the diet and the incidence of colonic carcinoma. Burkitt's postulate of a protective value of high fibre intake receives backing from 3 epidemiological studies from Israel, Scandinavia, and San Francisco.
D002784:Cholesterol / Q000032:analysis; D003110:Colonic Neoplasms / Q000209:etiology*; D004247:DNA / Q000096:biosynthesis; D014676:Diet, Vegetarian; D004041:Dietary Fats / Q000378:metabolism*; D004043:Dietary Fiber; D005243:Feces / Q000032:analysis / Q000382:microbiology; D005966:Glucuronidase / Q000378:metabolism; D006801:Humans; D007413:Intestinal Mucosa / Q000378:metabolism; D012004:Rectal Neoplasms / Q000209:etiology*
D004041:Dietary Fats; D004043:Dietary Fiber; D004247:DNA; D002784:Cholesterol; D005966:Glucuronidase
Zur Atiopathogenese des kolorektalen Karzinoms.